Traditional oriental medicine isn’t commonly used in PCT; but that doesn’t mean it doesn’t have some application. In fact, a supplement known as Deer Antler Velvet is known to contain proteins that stimulate muscle growth - this can therefore be used alongside your hormone regulators and supplements to ensure that you don’t lose too much of your muscle mass, weight or strength during PCT. Deer Antler Velvet won’t help testosterone production per se but it might make it easier for you to acclimatise to PCT.
When it comes to PCT, one of the most efficient methods to accomplishing these things is to obtain a SERM (Selective Estrogen Receptor Modulator). A SERM replaces estrogen in its receptor, preventing cell attachment, which causes the displaced hormone to eventually die out. After a cycle is over, the level of androgens in the body drop drastically. The body compensates with an overproduction of estrogen to keep steroid levels up. Estrogen as well inhibits the production of natural testosterone, and in the period between the return of natural testosterone and the end of a cycle, a lot of mass is lost. So its in everybody’s best interest to bring back natural test as soon as humanly possible. Clomid and Nolvadex will reduce the post-cycle estrogen, so that a steroid deficiency is constated and the hypothalamus is stimulated to regenerate natural testosterone production in the body. That’s basically how the mechanism works, nothing more, nothing less.
It was once commonly used during PCT in the belief it will aid testosterone restoration, however this is flawed due to its mechanism of action. The drug mimics the effects of LH in the body, stimulating the Leydig cells to produce testosterone in the testes . This can be fruitful in rectify existing, or avoiding testicular atrophy on cycle. It will not aid the process of recovery in the post cycle phase however, as the drug will bring about heightened oestrogen levels due to the greater aromatising of the testosterone being produced in the testes , thus bringing about greater inhibition of the HPTA .