It is of particular importance to evaluate patients thoroughly for possible ARDS and AE-IPF, since such patients may mimic AIP. Although not only the clinical manifestation, but DAD features in histology of ARDS is similar to AIP, in contrast to the idiopathy of AIP, [ 4 ] the indispensable cause must be present in ARDS. Also, the fibrosis in AIP has its peculiarity which is active and proliferative with minimal deposition of collagen. However, some researchers propose AIP as a possible cause or subtype of ARDS for their high similarities that is still controversial [ 8 , 9 ]. Whilst, AE-IPF, characterized by rapid deterioration at any point in the course of the disease, which is not secondary to infection, pulmonary embolism, or heart failure [ 10 , 11 ], is an acute insult to the lung over and above the underlying UIP. In short, the different etiology and HRCT feature are the critical points for antidiastole AIP from AE-IPF.
Support and advocacy groups can help you connect with other patients and families, and they can provide valuable services. Many develop patient-centered information and are the driving force behind research for better treatments and possible cures. They can direct you to research, resources, and services. Many organizations also have experts who serve as medical advisors or provide lists of doctors/clinics. Visit the group’s website or contact them to learn about the services they offer. Inclusion on this list is not an endorsement by GARD.